[Latin Name]Huperzia serratum

[Source] Huperziceae whole herb from China

[Appearance]Brown to white

[Ingredient]Huperzine A

[Specification]Huperzine A 1% – 5%, HPLC

[Solubility] Soluble in chloroform, methanol, ethanol, slightly soluble in water

[Particle size] 80 Mesh

[Loss on drying] ≤5.0%

[Heavy Metal] ≤10PPM

[Pesticide residue] EC396-2005, USP 34, EP 8.0, FDA

[Storage] Store in cool & dry area, keep away from the direct light and heat.

[Shelf life] 24 Months

[Package] Packed in paper-drums and two plastic-bags inside.

[What is Huperzine A]

Huperzia is a type of moss that grows in China. It is related to club mosses (the Lycopodiaceae family) and is known to some botanists as Lycopodium serratum . The whole prepared moss was used traditionally. Modern herbal preparations use only the isolated alkaloid known as huperzine A. Huperzine A is an alkaloid found in huperzia that has been reported to prevent the breakdown of acetylcholine, an important substance needed by the nervous system to transmit information from cell to cell. Animal research has suggested that huperzine A’s ability to preserve acetylcholine may be greater than that of some prescription drugs. Loss of acetylcholine function is a primary feature of several disorders of brain function, including Alzheimer’s disease . Huperzine A may also have a protective effect on brain tissue, further increasing its theoretical potential for helping reduce symptoms of some brain disorders.

[Function] Used in alternative medicine, huperzine A has been found to act as a cholinesterase inhibitor, a type of medicine used to prevent the breakdown of acetylcholine (a chemical essential to learning and memory).

Not only used as a treatment for Alzheimer’s disease, huperzine A is also said to enhance learning and memory and to protect against age-related cognitive decline.

In addition, huperzine A is sometimes used to boost energy, increase alertness, and aid in the treatment of myasthenia gravis (an autoimmune disorder that affects the muscles).

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Apitherapy is the medical use of honey bee products. This can include the use of honey, pollen, bee bread, propolis, royal jelly and bee venom.
Most claims of apitherapy have not been proved to the scientific standards of evidence-based medicine and are anecdotal in nature. A wide variety of conditions and diseases have been suggested by believers in the therapy as candidates for it, the most well-known being bee venom therapy for autoimmune diseases and multiple sclerosis.
The exact origins of apitherapy are difficult to pinpoint and can be traced back, in a general sense, to ancient Egypt, Greece, and China. Use of honey and other bee products can be traced back thousands of years and healing properties are included in many religious texts including the Veda, Bible, and Quran. These are mostly attributed to nutritional benefits of consumption of bee-products and not use of bee venom.

The more modern study of apitherapy, specifically bee venom, was initiated through the efforts of Austrian physician Phillip Terc in his published results “Report about a Peculiar Connection Between the Bee stings and Rheumatism” in 1888. More recent popularity can be drawn to Charles Mraz (1905–1999) a beekeeper from Vermont, United States over the past 60 years
Apitoxin, or honey bee venom, is a bitter colourless liquid; its active portion a mixture of proteins, which causes local inflammation and acts as an anticoagulant. A honeybee can inject 0.1 mg of venom via its stinger. It may have similarities to sea nettle toxin.
The main component is melittin amounting to 52% of venom peptides.
Apamin increases cortisol production in the adrenal gland. Apamin is a mild neurotoxin.
Adolapin,[3] contributing 2–5% of the peptides, acts as an anti-inflammatory and analgesic because it blocks cyclooxygenase.
Phospholipase A2 amounts to 10–12% of peptides and it is the most destructive component of apitoxin. It is an enzyme which degrades the phospholipids which cellular membranes are made of. It also causes decreased blood pressure and inhibits blood coagulation. Phospholipase A2 activates arachidonic acid which is metabolized in the cyclooxygenase-cycle to form prostaglandins. Prostaglandins regulate the body’s inflammatory response.
Hyaluronidase contributing 1–3% of peptides dilates the capillaries causing the spread of inflammation.
Histamine contributing 0.5–2% and is involved in the allergic response.
Dopamine and noradrenaline which contribute 1–2% increase pulse rate.
Protease-inhibitors contribute 2% and act as anti-inflammatory agents and stop bleeding.
Tertiapin.
More info at https://en.wikipedia.org/wiki/Apitoxin

Juan Gonzalo Angel
www.tvagro.tv

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