[Latin Name] Camellia sinensis

[Plant Source] China

[Specifications]

Total tea polyphenols 40%-98%

Total catechins 20%-90%

EGCG 8%-60%

[Appearance] Yellow brown powder

[Plant Part Used] Green tea leaf

[Particle size] 80 Mesh

[Loss on drying] ≤5.0%

[Heavy Metal] ≤10PPM

[Storage] Store in cool & dry area, keep away from the direct light and heat.

[Package] Packed in paper-drums and two plastic-bags inside.

[What is green tea extract]

Green tea is the second largest beverage demanded by consumers worldwide. Used in China and India for its medicinal effects. There are several compounds extracted from green tea including catechins which contain an enormous amount of hydroxyphenols that are easily oxidized, congregated and contracted, which explains its good anti-oxidation effect. Its anti-oxidation effect is 25-100 times as strong as those of vitamin C and E.

It is widely used in medicines, agriculture, and chemical and food industries. This extract prevents cardio-vascular disease, lowers the risk of cancer, and decreases blood sugar and blood pressure, as well as viruses. In the food industry, the anti-oxidation agent used for preserving food and cooking oils.

[Function]

1. Green tea extract can reduce blood pressure, blood sugar, blood lipids.

2. Green tea extract has the function of removing radicals and anti-aging.

3. Green tea extract can enhance the immune function and prevention of colds.

4. Green tea extract will anti-radiation,anti-cancer, inhibiting the increasing of cancer cell.

5. Green tea extract used to anti-bacterium, with the function of sterilization and deodorization.

[Application]

1.Applied in cosmetics field, Green tea extract owns the effect of anti-wrinkle and anti-Aging.

2.Applied in food field, Green tea extract is used as natural antioxidant, antistaling agent, and anti-fading agents.

3.Applied in pharmaceutical field, Green tea extract is used to prevent and cure cardiovascular disease, diabetes.

Warning: Long, science-heavy video. No funny cats or guys getting hit in the nuts.

Artificial sweeteners, or as they are usually referred to in the literature “non-caloric” sweeteners or “high-intensity” sweeteners, address the problem of sugar calorie excess in our diet. They’ve been demonstrated to support weight loss or maintenance, reduce cavities and can be part of a healthy lifestyle.

Much focus has been on

CSPI report: https://www.cspinet.org/reports/chemcuisine.htm
I disagree with CSPI on about 25% of their rankings, but I appreciate that they take a very conservative stance.

Citations:
Aspartame:
1. Comp Funct Genomics. 2010. In vivo cytogenetic studies on aspartame.
2. Drug Chem Toxicol. 2004 Aug;27(3):257-68. Genotoxicity of aspartame.
3. Am J Ind Med. 2010 Dec;53(12):1197-206. Aspartame administered in feed, beginning prenatally through life span, induces cancers of the liver and lung in male Swiss mice.
4. Toxicol In Vitro. 2011 Feb;25(1):286-93. In vitro effect of aspartame in angiogenesis induction.

Sucralose:
5. Regul Toxicol Pharmacol. 2009 Oct;55(1):1-5. An overview of the safety of sucralose.
6. Regul Toxicol Pharmacol. 2009 Oct;55(1):6-12. Expert panel report on a study of Splenda in male rats.
7. Food Chem Toxicol. 2000;38 Suppl 2:S53-69. Acute and subchronic toxicity of sucralose.
8. Food Chem Toxicol. 2000;38 Suppl 2:S71-89. A combined chronic toxicity/carcinogenicity study of sucralose in Sprague-Dawley rats.
9. Food Chem Toxicol. 2000;38 Suppl 2:S91-7. A carcinogenicity study of sucralose in the CD-1 mouse.

AceK:
10. Horm Metab Res. 1987 Jun;19(6):233-8. The effect of artificial sweetener on insulin secretion. 1. The effect of acesulfame K on insulin secretion in the rat (studies in vivo).
11. Food Chem Toxicol. 1997 Dec;35(12):1177-9. In vivo cytogenetic studies on mice exposed to acesulfame-K–a non-nutritive sweetener.

General reviews:
12. Ann Oncol. 2004 Oct;15(10):1460-5. Artificial sweeteners–do they bear a carcinogenic risk?
13. Yale J Biol Med. 2010 Jun;83(2):101-8. Gain weight by “going diet?” Artificial sweeteners and the neurobiology of sugar cravings
14. Int J Obes Relat Metab Disord. 1996 Mar;20 Suppl 2:S12-7. Effect of sucrose and sweeteners on appetite and energy intake.
15. Am J Clin Nutr. 2009 Jan;89(1):1-14. Nonnutritive sweetener consumption in humans: effects on appetite and food intake and their putative mechanisms.
16. Physiol Behav. 2010 Apr 26;100(1):55-62. High-intensity sweeteners and energy balance.
17. Physiol Behav. 2009 Dec 7;98(5):618-24. Effect of moderate intake of sweeteners on metabolic health in the rat.
18. Food Addit Contam. 2006 Apr;23(4):327-38. The intake of intense sweeteners – an update review.