[Latin Name] Haematococcus Pluvialis

[Plant Source] from China

[Specifications]1% 2% 3% 5%

[Appearance] Dark red Powder

[Particle size] 80 Mesh

[Loss on drying] ≤5.0%

[Heavy Metal] ≤10PPM

[Storage] Store in cool & dry area, keep away from the direct light and heat.

[Shelf life] 24 Months

[Package] Packed in paper-drums and two plastic-bags inside.

[Net weight] 25kgs/drum

Brief Introduction

Astaxanthin is a natural nutritional component, it can be found as a food supplement. The supplement is intended for human, animal, and aquaculture consumption.

Astaxanthin is a carotenoid. It belongs to a larger class of phytochemicals known as terpenes, which are built from five carbon precursors; isopentenyl diphosphate and dimethylallyl diphosphate . Astaxanthin is classified as a xanthophyll (originally derived from a word meaning “yellow leaves” since yellow plant leaf pigments were the first recognized of the xanthophyll family of carotenoids), but currently employed to describe carotenoid compounds that have oxygen-containing moities, hydroxyl or ketone , such as zeaxanthin and canthaxanthin. Indeed, astaxanthin is a metabolite of zeaxanthin and/or canthaxanthin, containing both hydroxyl and ketone functional groups. Like many carotenoids, astaxanthin is a colorful, lipid-soluble pigment. This colour is due to the extended chain of conjugated (alternating double and single) double bonds at the centre of the compound. This chain of conjugated double bonds is also responsible for the antioxidant function of astaxanthin (as well as other carotenoids) as it results in a region of decentralized electrons that can be donated to reduce a reactive oxidizing molecule.

Function:

1.Astaxanthin is a powerful antioxidant and may protect against oxidative damage to body tissues.

2.Astaxanthin can improve the immune response by increasing the number of antibody producing cells.

3.Astaxanthin is a potential candidate to treat neurodegenerative disease such as Alzhimer and Parkinson diease.

4.Astaxanthin dan reduce UVA-light damage to skin such as sunburn, inflammation, ageing and skin cancer.

Application

1.When applied in pharmaceutical field, astaxanthin powder has the good function of antineoplastic;

2.When applied in health food field, astaxanthin powder is used as food additives for pigment and health care;

3.When applied in cosmetic field, astaxanthin powder has the good function of antioxidant and anti-aging;

4.When applied in animal feeds field, astaxanthin powder is used as animal feed additive to impart coloration, including farm-raised salmon and egg yolks.

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Warning: Long, science-heavy video. No funny cats or guys getting hit in the nuts.

Artificial sweeteners, or as they are usually referred to in the literature “non-caloric” sweeteners or “high-intensity” sweeteners, address the problem of sugar calorie excess in our diet. They’ve been demonstrated to support weight loss or maintenance, reduce cavities and can be part of a healthy lifestyle.

Much focus has been on

CSPI report: https://www.cspinet.org/reports/chemcuisine.htm
I disagree with CSPI on about 25% of their rankings, but I appreciate that they take a very conservative stance.

Citations:
Aspartame:
1. Comp Funct Genomics. 2010. In vivo cytogenetic studies on aspartame.
2. Drug Chem Toxicol. 2004 Aug;27(3):257-68. Genotoxicity of aspartame.
3. Am J Ind Med. 2010 Dec;53(12):1197-206. Aspartame administered in feed, beginning prenatally through life span, induces cancers of the liver and lung in male Swiss mice.
4. Toxicol In Vitro. 2011 Feb;25(1):286-93. In vitro effect of aspartame in angiogenesis induction.

Sucralose:
5. Regul Toxicol Pharmacol. 2009 Oct;55(1):1-5. An overview of the safety of sucralose.
6. Regul Toxicol Pharmacol. 2009 Oct;55(1):6-12. Expert panel report on a study of Splenda in male rats.
7. Food Chem Toxicol. 2000;38 Suppl 2:S53-69. Acute and subchronic toxicity of sucralose.
8. Food Chem Toxicol. 2000;38 Suppl 2:S71-89. A combined chronic toxicity/carcinogenicity study of sucralose in Sprague-Dawley rats.
9. Food Chem Toxicol. 2000;38 Suppl 2:S91-7. A carcinogenicity study of sucralose in the CD-1 mouse.

AceK:
10. Horm Metab Res. 1987 Jun;19(6):233-8. The effect of artificial sweetener on insulin secretion. 1. The effect of acesulfame K on insulin secretion in the rat (studies in vivo).
11. Food Chem Toxicol. 1997 Dec;35(12):1177-9. In vivo cytogenetic studies on mice exposed to acesulfame-K–a non-nutritive sweetener.

General reviews:
12. Ann Oncol. 2004 Oct;15(10):1460-5. Artificial sweeteners–do they bear a carcinogenic risk?
13. Yale J Biol Med. 2010 Jun;83(2):101-8. Gain weight by “going diet?” Artificial sweeteners and the neurobiology of sugar cravings
14. Int J Obes Relat Metab Disord. 1996 Mar;20 Suppl 2:S12-7. Effect of sucrose and sweeteners on appetite and energy intake.
15. Am J Clin Nutr. 2009 Jan;89(1):1-14. Nonnutritive sweetener consumption in humans: effects on appetite and food intake and their putative mechanisms.
16. Physiol Behav. 2010 Apr 26;100(1):55-62. High-intensity sweeteners and energy balance.
17. Physiol Behav. 2009 Dec 7;98(5):618-24. Effect of moderate intake of sweeteners on metabolic health in the rat.
18. Food Addit Contam. 2006 Apr;23(4):327-38. The intake of intense sweeteners – an update review.